Et oui sans haine, ni violence. Comme Bébert le Niçois

Therefore, it may be tempting to try to link the affinity of ChAdOx1 for CAR with platelet aggregation and TTS. However, previous studies demonstrate that adenovirus-platelet aggregates are rapidly trafficked to the liver where they are sequestered by Kupffer cells and degraded (28). While it was possible to induce thrombocytopenia in mice following an intravenous dose of 1011 VP (Viral Particles)/ml replication-deficient adenoviral particles (12) (a dose ~7500× higher than that given, intramuscularly, in the ChAdOx1 nCoV-19 vaccine, assuming a 20-g mouse and a 75-kg human), this did not result in any thrombotic events. Another study in Rhesus macaques observed the opposite effect, longer clotting times, presumably as a result of the diminished platelet count and/or depletion of circulating FX (29, 30). Therefore, we believe it is unlikely that direct association between ChAdOx1 and platelets drives thrombotic events, regardless of their CAR expression status, in TTS.

D'autant plus que la France doit être aujourd'hui un des seuls si ce n'est le seul pays à ne pas avoir réintégré les suspendus (soignants, pompiers, etc...)

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